SODERBERGD
Active member
Concur . . .
Yes. For those of you that don't know the story, the abridged version is; Walter had a male red bloodred (now at Bayou) that had the most white on the sides I’d seen up to that time (4-5 years ago). He got it from McDonald. I told him white’s been on the sides of some bloods since their induction into Corndom, but I loaned him a female of mine that had a little white on her sides (conjunct to ventor). There were no p/s snakes among the F1s. Walter didn’t keep his F1s of the co-op project, but when I bred two of the F1 females to two of their brothers, voila. More than half of them had obscene amounts of white on their sides. Oh yeah, bred one of the F1s boys back to the female of mine (his mother) that Walter borrowed, and got around 50% p/s bloods. Rob can say what happened after that, but as far as we know, the F1s I had from that pairing were the only ones available. Anyone that bought bloods from Walter that year, probably has the “trait potential”, but may not know it. Heck, I also sold some of mine as regular bloods. Keep in mind that the matriarch of these (my female blood) had been a primary breeder of mine for years, and I’d been selling dozens of her babies up to six years before that. No telling how many people have the “trait”, and don’t know it. SOOOO, since I bred the mother back to her son and got phenotypes, whatever is going on, seemed to at least be linked to my female (also in light of none of the F1s being p/s). Was the McDonald animal a different gene/trait? It didn’t appear to be the same genetics mine had. To skip a generation like that certainly suggests recessive heritability, but one more generation of trials should clear that up. I think KJ is also doing the same trials this year, so that’s at least three of us that will have F2 outcross and F3 phenotypic evidence.
Yes. For those of you that don't know the story, the abridged version is; Walter had a male red bloodred (now at Bayou) that had the most white on the sides I’d seen up to that time (4-5 years ago). He got it from McDonald. I told him white’s been on the sides of some bloods since their induction into Corndom, but I loaned him a female of mine that had a little white on her sides (conjunct to ventor). There were no p/s snakes among the F1s. Walter didn’t keep his F1s of the co-op project, but when I bred two of the F1 females to two of their brothers, voila. More than half of them had obscene amounts of white on their sides. Oh yeah, bred one of the F1s boys back to the female of mine (his mother) that Walter borrowed, and got around 50% p/s bloods. Rob can say what happened after that, but as far as we know, the F1s I had from that pairing were the only ones available. Anyone that bought bloods from Walter that year, probably has the “trait potential”, but may not know it. Heck, I also sold some of mine as regular bloods. Keep in mind that the matriarch of these (my female blood) had been a primary breeder of mine for years, and I’d been selling dozens of her babies up to six years before that. No telling how many people have the “trait”, and don’t know it. SOOOO, since I bred the mother back to her son and got phenotypes, whatever is going on, seemed to at least be linked to my female (also in light of none of the F1s being p/s). Was the McDonald animal a different gene/trait? It didn’t appear to be the same genetics mine had. To skip a generation like that certainly suggests recessive heritability, but one more generation of trials should clear that up. I think KJ is also doing the same trials this year, so that’s at least three of us that will have F2 outcross and F3 phenotypic evidence.
), I'll call/label them p/s bloodreds.
